[1]汪沙 林斯丽 综述 段华**审校.趋化因子在子宫内膜异位症发病机制中作用的研究进展[J].中国微创外科杂志,2020,01(7):644-648.
点击复制

趋化因子在子宫内膜异位症发病机制中作用的研究进展()
分享到：

《中国微创外科杂志》[ISSN:1009-6604/CN:11-4526/R]

卷:
01
期数:
2020年7期
页码:
644-648
栏目:
文献综述
出版日期:
2020-09-15

文章信息/Info

作者:
汪沙 林斯丽 综述 段华**审校
(首都医科大学附属北京妇产医院妇科微创中心，北京100006)
文献标志码:
A

参考文献/References:

[1]韩肖彤,郭红燕.口服避孕药预防子宫内膜异位症的研究进展.中国微创外科杂志,2018,18(4):341-344.
[2]Wickstrm K, StavréusEvers A, Vercauteren O, et al. Effect of lignocaine on IL6, IL8, and MCP1 in peritoneal macrophages and endometriotic stromal cells. Reprod Sci,2017,24(3): 382-392.
[3]Hanada T, Tsuji S, Nakayama M, et al. Suppressive regulatory T cells and latent transforming growth factorβexpressing macrophages are altered in the peritoneal fluid of patients with endometriosis. Reprod Biol Endocrinol,2018,16(1):9-17.
[4]Tanaka Y， Mori T， Ito F， et al. Exacerbation of endometriosis due to regulatory T cell dysfunction. J Clin Endocrinol Metab,2017,102(9):3206-3217.
[5]Borrelli GM, Abro MS, Taube ET, et al. Immunohistochemical investigation of metastasisrelated chemokines in deepinfiltrating endometriosis and compromised pelvic sentinel lymph nodes. Reprod Sci,2015,22(12):1632-1642.
[6]Mlu �瘙 塅 an AM, Drugan T, Ciortea R, et al. Endometriosisassociated changes in serum levels of interferons and chemokines. Turk J Med Sci,2017,47(1):115-122.
[7]Jin CH, Yi KW, Ha YR, et al. Chemerin expression in the peritoneal fluid, serum, and ovarian endometrioma of women with endometriosis. Am J Reprod Immunol,2015,74(4):379-386.
[8]Kayisli UA, Mahutte NG, Arici A. Uterine chemokines in reproductive physiology and pathology. Am J Reprod Immunol,2015,47(4):213-221.
[9]Hornung D, Ryan IP, Chao VA, et al. Immunolocalization and regulation of the chemokine RANTES in human endometrial and endometriosis tissues and cells. J Clin Endocrinol Metab,1997,82(5):1621-1628.
[10]王悦,张丽珠,王秀云.子宫内膜异位症患者腹腔液及血清中趋化因子RANTES的含量及意义.中华妇产科杂志,2001,36(8):496-497.
[11]周夏伶,何伟,王久兰,等.血清与腹腔液中趋化因子RANTES水平在子宫内膜异位症患者中的临床应用.现代生物医学进展,2015,15(16):3119-3121.
[12]胡倩倩,严浩,李盛.VEGF、RANTES、sflt1及MCP1在子宫内膜异位症合并代谢综合征的表达及意义.中国妇幼健康研究,2016,27(4):463-465.
[13]Ku P, Charkiewicz R, KlasaMazurkiewicz D, et al. Profiling of selected angiogenesisrelated genes in serous ovarian cancer patients. Adv Med Sci,2017,62(1):116-120.
[14]Mihalyi A, Gevaert O, Kyama CM， et al. Noninvasive diagnosis of endometriosis based on a combined analysis of six plasma biomarkers. Hum Reprod,2010,25(3):654-664.
[15]Iwabe T, Harada T, Tsudo T, et al. Pathogenetic significance of increased levels of interleukin8 in the peritoneal fluid of patients with endometriosis. Fertil Steril,1998, 69(5):924-930.
[16]CalhazJorge C, Costa AP, Santos MC, et al. Peritoneal fluid concentrations of interleukin8 in patients with endometriosis depend on the severity of the disorder and are higher in the luteal phase. Hum Reprod,2003,18(3):593-597.
[17]Sikora J, SmyczKubańska M, MielczarekPalacz A, et al. Abnormal peritoneal regulation of chemokine activationThe role of IL8 in pathogenesis of endometriosis. Am J Reprod Immunol,2017,77(4):e12622.
[18]Makabe T, Koga K, Miyashita M, et al. Drospirenone reduces inflammatory cytokines, vascular endothelial growth factor (VEGF) and nerve growth factor (NGF) expression in human endometriotic stromal cells. J Reprod Immunol,2017,119(Complete):44-48.
[19]Lin J， Gu Y. Effect of monocyte chemoattractant protein1 and estradiol on the secretion of vascular endothelial growth factor in endometrial stromal cells in vitro. Fertil Steril,2005,84(6):1793-1796.
[20]Park JY， Kang YW， Choi BY， et al. CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs. J Recept Signal Transduct Res,2017,37(4):416-421.
[21]Hornung D. Localization in tissues and secretion of eotaxin by cells from normal endometrium and endometriosis. J Clin Endocrinol Metab,2000, 85(7):2604-2608.
[22]Salcedo R, Young HA, Ponce ML, et al. Eotaxin (CCL11) induces in vivo angiogenic responses by human CCR3+ endothelial cells. J Immunol,2001,166(12):7571-7578.
[23]Ouyang Z, Osuga Y, Hirota Y, et al. Interleukin4 induces expression of eotaxin in endometriotic stromal cells. Reprod Sci,2010,94(1):58-62.
[24]Zhou WJ, Yang HL, Shao J, et al. Antiinflammatory cytokines in endometriosis. Cell Mol Life Sci,2019,76(11):2111-2132.
[25]陈惠娴,张真真,万贵平.子宫内膜异位患者CXCL16的表达情况及诊断价值.中国医药导报,2018,15(15):92-95.
[26]Mulayim N, Savlu A, GuzelogluKayisli O, et al. Regulation of endometrial stromal cell matrix metalloproteinase activity and invasiveness by interleukin8. Fertil Steril,2004,81(Suppl 1):S904-S911.
[27]Li MQ, Li HP, Meng YH, et al. Chemokine CCL2 enhances survival and invasiveness of endometrial stromal cells in an autocrine manner by activating Akt and MAPK/Erk1/2 signal pathway. Fertil Steril,2012,97(4):919-929.e1.
[28]Berkkanoglu M, Arici A. Immunology and endometriosis. Am J Reprod Immunol,2015,50(1):48-59.
[29]Takamura M, Koga K, Izumi G, et al. Simultaneous detection and evaluation of four subsets of CD4+ T lymphocyte in lesions and peripheral blood in endometriosis. Am J Reprod Immunol,2015,74(6):480-486.
[30]罗雪珍,史颖莉,王凌,等.CCR4及CCR8在子宫内膜异位症患者在位内膜及异位灶的表达. 现代妇产科进展,2011,20(1):39-41.
[31]张达,杜艳辉,袁芳.子宫内膜异位症患者免疫调节Th17细胞及Treg细胞的表达意义.中华细胞与干细胞杂志(电子版),2019,9(5):292-297.
[32]Chang KK, Liu LB, Jin LP, et al. IL27 triggers IL10 production in Th17 cells via a cMaf/RORγt/Blimp1 signal to promote the progression of endometriosis. Cell Death Dis,2017,8(3):e2666.
[33]Li MZ, Wu YH, Ali M, et al. Endometrial stromal cells treated by tumor necrosis factorα stimulate macrophages polarized toward M2 via interleukin6 and monocyte chemoattractant protein1. J Obstet Gynaecol Res,2020,46(2):293-301.
[34]Li MQ, Luo XZ, Meng YH, et al. CXCL8 enhances proliferation and growth and reduces apoptosis in endometrial stromal cells in an autocrine manner via a CXCR1triggered PTEN/AKT signal pathway. Hum Reprod,2012,27(7):2107-2116.
[35]Li MQ, Li HP, Meng YH, et al. Chemokine CCL2 enhances survival and invasiveness of endometrial stromal cells in an autocrine manner by activating Akt and MAPK/Erk1/2 signal pathway. Reprod Sci,2012,97(4):919-929.
[36]VallvéJuanico J, Houshdaran S, Giudice LC. The endometrial immune environment of women with endometriosis. Hum Reprod Update,2019,25(5):564-591.
[37]Zhou WJ, Hou XX, Wang XQ, et al. The CCL17CCR4 axis between endometrial stromal cells and macrophages contributes to the high levels of IL6 in ectopic milieu. Am J Reprod Immunol,2017,78(2): e12644.

备注/Memo

备注/Memo:
基金项目：国家自然科学基金（81571412）；首都医科大学附属北京妇产医院中青年学科骨干培养专项（FCYY201920）**通讯作者，Email:duanhuasci@163.com
更新日期/Last Update: 2020-10-13