[1]王海霞,朱曦,修典荣.危重疾病肌肉恶病质的研究进展[J].中国微创外科杂志,2006,06(12):998-1000.
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危重疾病肌肉恶病质的研究进展()
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《中国微创外科杂志》[ISSN:1009-6604/CN:11-4526/R]

卷:
06
期数:
2006年12期
页码:
998-1000
栏目:
文献综述
出版日期:
2006-12-30

文章信息/Info

作者:
王海霞朱曦修典荣
北京大学第三医院危重医学科,北京,100083
分类号:
R746
文献标志码:
A

参考文献/References:

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[4]Lacomis D,Zochodne DW,Bird SJ.Critical illness myopathy.Muscle Nerve,2000,23:1785-1788.
[5]项建斌.肌肉恶液质细胞内机制与分子调节的现代观念.国外医学?外科学分册,2001,28(4):233-234.
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[10]Friedrich O,Rainer H.Nutritional consequences of critical illness myopathies.J Nutr,2005,135:1813s-1817s.
[11]姚咏明,柴家科,林洪远,主编.现代脓毒症理论与实践.北京:科学出版社,2005.471.
[12]Hobler SC,Tiao G,Fischer JE,et al.The sepsis-induced increase in muscle proteolysis is blocked by specific proteasome inhibitors.Am J Physiol,1998,272:R30-R37.
[13]柴家科,吴焱秋,盛志勇,等.脓毒症对大鼠骨骼肌组织泛素及泛素化蛋白表达的影响.中华外科杂志,2001,39:721-723.
[14]Bolton CF.Sepsis and the systemic inflammatory response syndrome:neuromuscular manifestations.Crit Care Med,1996,24:1408-1416.
[15]Mitch W,Goldberg A.Mechanisms of muscle wasting.N Engl J Med,1996,335:1897-1905.
[16]Cooney RN,Kimball SR,Vary TC.Regulation of skeletal muscle protein turnover during sepsis(mechanisms and mediators).Shock,1997,7:1-16.
[17]Herridge M,Cheung A,Tansey C,et al.A one year outcomes in survivors of the acute respiratory distress syndrome.N Engl J Med,2003,348:683-693.
[18]吴焱秋,柴家科,盛志勇,等.糖皮质激素在烧伤大鼠骨骼肌蛋白降解中的作用及其与蛋白酶体亚基表达的关系.中华实验外科杂志,2003,20:211-213.
[19]Hasselgren PO.Molecular regulation of muscle cachexia:it may be more than the proteasome.Biochem Biophys Res Commun,2002,290:1-10.
[20]Sun Xiaoyan,Fischer DR.Expression and binding acivity of the glucocorticoid receptor are upregulated in septic muscle.Am J Physiol Regul Intergr Comp Physiol,2002,282:509-518.
[21]Penner G,Gang G.C/EBP DNA-binding activity is upregulated by a glucocorticoid dependent mechanism in sepic muscle.Am J Phsiol Regul Integr Comp Physiol,2002,282:439-444.
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更新日期/Last Update: 2014-03-03